ITEM#-100: Skin Disease 02
Stabilization Criteria
ITEM#-100 cannot be isolated using standard synthetic fabric filters. Active biological hosts exhibiting spreading pigmentation anomalies must be permanently maintained within a negative-pressure, hermetically sealed bio-hazard chamber within Sector-04. The internal atmosphere must undergo continuous chemical incineration to eliminate shed microscopic dermal particulates.
Medical personnel interacting with infected hosts are strictly required to wear heavy-grade pressurized hazmat plating with independent oxygen reserves. Direct tactile contact with an infected subject's skin barrier is an absolute contamination hazard. Any researcher displaying sudden, localized shifts in baseline epidermal coloration must face immediate, permanent quarantine separation.
Description
ITEM#-100: An aggressive, self-replicating bio-anomalous contagion of unverified molecular lineage. The phenomenon operates as an invasive cellular vector that systematically re-engineers the baseline pigment parameters of human epidermal tissue upon direct contact. The anomaly is classified as LABILIS due to its absolute immunity to traditional chemical sedatives, radiation purges, and targeted dermatological counter-measures.
The progression of the anomaly triggers upon initial exposure. During Phase 1, the host experiences an aggressive, erratic shifting of their natural skin tone. The contagion causes large, map-like patches of the skin to completely lose their baseline pigmentation, leaving smooth, distinct contrasts across the extremities. While early stages remain physically painless and show no immediate material decay, the rewritten cellular zones are entirely non-reactive to localized nerve stimuli.
If the contagion remains on the host over an extended timeline, the underlying biological mechanisms become highly lethal. During Phase 3, the altered patches begin to systematically compromise the host's vascular integrity, replacing natural subcutaneous tissue with a non-organic, synthetic lipid layer. Once the alteration reaches the primary circulatory system, it halts baseline respiratory oxygen transport, leading to internal asphyxiation. Dr. Lizzy Vance has ordered strict surveillance over the isolation ward to ensure no airborne cell flakes enter the facility's main ventilation system.